Flaminia Malvezzi

Flaminia Malvezzi Campeggi obtained the degree in Physics cum laude in 1997 from the University of Rome La Sapienza and the PhD, major Biophysics, at the University of Tor Vergata (Rome) in 2001. She performed part of her doctoral work at Max Planck Institute for Biophysical Chemistry in Goettingen (Germany). Her work was concentrated in analysing the proteins’ folding and unfolding through fluorescence spectroscopy and in studying the fluorescence emitted by the newly discovered red fluorescent protein DsRed. Her final report was published in Biophysical Journal and Biochemical and Biophysical Research Communication. In 2001 she moved to Verona (Italy), where she spent 5 years as postdoctoral researcher. She was involved in studying the role of Fourier Transform Infrared (FT-IR) Microspectroscopy and analysing the UV long term effects on human cells. In the meantime, she had several teaching assignments both at University and at second level. She obtained the official qualification for teaching in 2009, from the University of Venice. In 2010 she moved to Dublin as Research Officer for the RCSI. She managed the CEMP Marie Curie postdoctoral programme for researchers, for which she also organised the leadership and management development programme week (LMDP). She moved back to Verona in 2012, where she is involved in:

  • serving as science writer and research support at the Italian Cystic Fibrosis (CF) Research Foundation. In this role, she writes monthly updates on the research activities performed by Italian CF researchers, working on the improvement of their visibility and citation index. Responsible for the FFC convention Proceedings.
  • teaching mathematics and physics at don Bosco High School both in Italian and English. Developing recognised international projects such as eTwinning projects. Serving as tutor and teacher at the University of Verona in the context of both teachers’ apprenticeships and entrance exam for the Faculty of Medicine.

Nuove prospettive di cura per la fibrosi cistica

Da più di qualche anno si è aperta una nuova prospettiva per la ricerca di una cura del difetto di base nei pazienti affetti da Fibrosi Cistica (FC), al punto che è capitato di leggere in qualche incauta comunicazione mediatica “trovata cura per la Fibrosi Cistica”. La malattia è dovuta a una mutazione genetica che provoca il malfunzionamento di una proteina che agisce come canale per il passaggio degli ioni cloro fuori e dentro la cellula. Il suo nome, dal gene che la produce, è proteina CFTR.

Vent'anni di ricerca italiana sulla fibrosi cistica festeggiati con Paul Quinton

Lo scienziato Paul Quinton nel suo laboratorio della University of California San Diego (2015).

Per i vent'anni della Fondazione Italiana per la Fibrosi Cistica, il 27 e 28 maggio, in un incontro con i volontari che si è tenuto alle porte di Verona, il direttore scientifico della fondazione Gianni Mastella ha ripercorso la lunga storia di una malattia a base genetica più comune di quanto si pensi. Basti dire che in Italia c’è un portatore sano ogni 25 persone circa, la maggioranza delle quali non sa di esserlo.